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Transcriptional Dependencies in Diffuse Intrinsic Pontine Glioma
Surya Nagaraja
, Nicholas A. Vitanza
, Pamelyn J. Woo
, Kathryn R. Taylor
, Fang Liu
, Lei Zhang
, Meng Li
, Wei Meng
, Anitha Ponnuswami
, Wenchao Sun
, Jie Ma
,
Esther Hulleman
, Joanna Wysocka
, Tomek Swigut
, Yujie Tang
, Michelle Monje
Research output
:
Contribution to journal
›
Article
›
peer-review
313
Citations (Scopus)
Overview
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Neuroscience
Histone H3
100%
Enhancer Region
100%
Potassium Channel
100%
Bromodomain
100%
Histone Deacetylase Inhibitor
100%
Receptor
100%
Biochemistry, Genetics and Molecular Biology
Enhancer Region
100%
Transcription
100%
Bromodomain
100%
Histone H3
100%
Potassium Channel Function
100%
Keyphrases
Diffuse Intrinsic Pontine Glioma
100%
CDK7
28%
Pediatric Cancer
14%
Synergize
14%
Therapeutic Potential
14%
Cell of Origin
14%
Vulnerability
14%
HDAC Inhibition
14%
Glioma Cells
14%
Histone H3
14%
Histone Deacetylase Inhibitor (HDACi)
14%
H3K27M
14%
Aberrancy
14%
Channel Function
14%
Tumor Viability
14%
Tumor Invasion
14%
Super-enhancer
14%
Inhibitor Therapy
14%
Bromodomain Inhibitors
14%
Eph Receptor
14%
Oligodendroglial
14%
Lineage Genes
14%
Receptor Signaling
14%
Pathobiology
14%
Potassium Channel
14%
Medicine and Dentistry
Pontine Glioma
100%
Neoplasm
14%
Pediatric Cancer
14%
Histone H3
14%
Glioma Cell
14%
Receptor
14%
Potassium Channel
14%
Enhancer Region
14%
Histone Deacetylase Inhibitor
14%
Pharmacology, Toxicology and Pharmaceutical Science
Pontine Glioma
100%
Neoplasm
14%
Malignant Neoplasm
14%
Receptor
14%
Histone H3
14%
Potassium Channel
14%
Histone Deacetylase Inhibitor
14%